Heeft u de diagnose MS? Er zijn veel MS patienten die zich lieten testen door een ILADS arts en Lyme bleken te hebben
As minocycline, tinidazole and hydroxychloroquine are reportedly
capable of destroying both the spirochaetal and cystic L-form of B.
burgdorferi found in MS brains.
Dr. Klinghardt stated in one of
our interviews that he's never had a single patient with Alzheimer's,
ALS, Parkinson's disease or multiple sclerosis who tested negative for
Borrelia.
Vincent Marshall is een onderzoeker die veronderstelde
dat MS een chronische ziekte van het centrale zenuwstelsel is welke
veroorzaakt wordt door de spirocheet Borrelia. Zijn artikel uit 1988
gepubliceerd in het blad Hypothesis verwees naar een eerder artikel uit
1954, waarin gekeken werd naar de bacteriële etiologie voor MS,
gebruikmakend van zilverkleuringen in zenuw leasies, en dierlijke
isolatie van de laesies veroorzakende bacterie. Marshall geeft in zijn
artikel aan dat de demyelinisatie van het zenuwstelsel behandeld kan
worden met hoge dosis antibiotica welke de bloed-hersenbariëre kunnen
passeren en het centrale zenuwustelsel kunnen bereiken alsook de
mogelijkheid biedt tot het maken van een vaccin.
Microbioloog Dr Hoekstra:
Dr. Hoekstra's mentor was Lida Holmes Mattman.
Lida Mattman heeft de Borrelia bacterie 50 jaar bestudeerd en is
genomineerd voor de Nobel prijs. Zij heeft veel ontdekt en bewezen wbt
de Borrelia bacterie.
A different bacteria, operating on similar
principles, seems to be the organic cause of multiple sclerosis, says
Dr. Hoekstra. It's tentative name--not yet widely accepted by other
microbiologists--is Borrelia mylophora, so named because its
characteristics seem to resemble those of Borrelia burgdorferi, the
bacteria believed responsible for Lyme disease.
In multiple
sclerosis, the myelin sheath covering nerves gets eaten away by the
immune system, explains Dr. Hoekstra. "That is exactly like the hunters'
torches setting fire to the forest. Most of the destruction of the
myelin sheath takes place from actions of the white blood cells and
their antibodies. But their primary target is not the myelin sheath at
all. It's the Borrelia mylophora bacteria, running around in the nervous
system.
B. mylophora has an extremely high affinity for the myelin sheath. It loves it."
Unfortunately, the myelin sheath sustains a lot of collateral damage as
the immune cells attempt to find and destroy the microbe, Dr. Hoekstra
says.
The work on identifying a possible microbial agent in multiple
sclerosis has been under way for many years, since 1913. One of the
difficulties is the microbiologist must be very patient, able to culture
a blood specimen and keep it uncontaminated for as long as nine months
before a positive bacterial identification can be made, explains Dr.
Hoekstra.
Both P. acnes and B. mylophora are examples of stealth
pathogens, of organisms with deficient cell walls, capable of acting
secretly in the body, creating disease, and hardly leaving a trace.
Pushing Away Their Wheelchairs
Once you understand the nature of the organism causing an autoimmune
disease, it's much easier to develop an effective strategy against it,
says Dr. Hoekstra. "We have a number of multiple sclerosis patients who
have pushed away their wheelchairs or thrown away their canes and are
walking now. And their vision has been restored," he adds.
Physicians use different strategies in dealing with the underlying
bacteria. Dr. Hoekstra cites the example of Phoebe, a 36-year-old woman
with MS. After B. mylophora was cultured from her blood, Phoebe's doctor
prescribed a heavy dose (about 100 mg daily) of a standard antibiotic
called doxycycline. After four weeks, she was able to lift both hands in
the air, comb her hair without losing her balance, see clearly again
without dizziness, and move about without her cane; after six months
(four of which involved continuous dosing), Phoebe was free of all symptoms, says Dr. Hoekstra.
The successful use of this antibiotic against B. mylophora was first
verified by a physician in South Dakota who reasoned that the symptoms
of MS (which he had) were suggestively similar to those of Lyme disease,
which responds fairly well to doxycycline. After dosing himself for
three months with the antibiotic, he was symptom free.
MS en Lyme
Het bleek dat er een significant aantal overlappende gevallen was van
schizofrenie, MS, en de verdeling van Ixodes ricinus teken die de ziekte
van Lyme in Europa verspreiden. Fritzsche observeerde dat gematigde
temperatuur klimaten een hoger aantal infecties had van teken besmet met
de Borrelia garinii, en dat dit infectie aantal overeen kwam met de
verdeling van MS gevallen.
Bovendien merkte Fritzsche op dat nieuwe
excessen van MS en die van schizofrenie negen maanden uit elkaar lagen.
Dit suggereerde dat ze verband hielden met de activiteit van de Ixodes
ricinus teek in Europa op het moment van zijn conceptie en geboorte. Ook
in de VS werden deze negen maanden clusters van MS en schizofrenie
gevonden wat de activiteit van de Ixodes pacificus en de Ixodus
scapularis geboortecyclus weerspiegelden. Beide zijn ziekte van Lyme
veroorzakende teken. Fritzsche spreekt hierin ook zijn bezorgdheid uit
over de mogelijke overdracht van de ziekte van Lyme van moeder op kind
tijdens de zwangerschap, alhoewel officiële richtlijnen nog steeds
volhouden dat de ziekte van Lyme niet besmettelijk is.
http://www.ncbi.nlm.nih.gov/pubmed/12537588
Alan MacDonald zegt: Alles wat Syfiles kan, kan de Borrelia bacterie ook!!
Apart from its devastating impact on individuals and their families,
multiple sclerosis (MS) creates a huge economic burden for society by
mainly afflicting young adults in their most productive years.
Although effective strategies for symptom management and disease
modifying therapies have evolved, there exists no curative treatment
yet.
Worldwide, MS prevalence parallels the distribution of the
Lyme disease pathogen Borrelia (B.) burgdorferi, and in America and
Europe, the birth excesses of those individuals who later in life
develop MS exactly mirror the seasonal distributions of Borrelia
transmitting Ixodes ticks.
In addition to known acute
infections, no other disease exhibits equally marked epidemiological
clusters by season and locality, nurturing the hope that prevention
might ultimately be attainable.
As minocycline, tinidazole and
hydroxychloroquine are reportedly capable of destroying both the
spirochaetal and cystic L-form of B. burgdorferi found in MS brains,
there emerges also new hope for those already afflicted.
The
immunomodulating anti-inflammatory potential of minocycline and
hydroxychloroquine may furthermore reduce the Jarisch Herxheimer
reaction triggered by decaying Borrelia at treatment initiation.
Even in those cases unrelated to B. burgdorferi, minocycline is known
for its beneficial effect on several factors considered to be
detrimental in MS. Patients receiving a combination of these
pharmaceuticals are thus expected to be cured or to have a longer period
of remission compared to untreated controls.
Although the goal of
this rational, cost-effective and potentially curative treatment seems
simple enough, the importance of a scientifically sound approach cannot
be overemphasised.
A randomised, prospective, double blinded
trial is necessary in patients from B. burgdorferi endemic areas with
established MS and/or Borrelia L-forms in their cerebrospinal fluid, and
to yield reasonable significance within due time, the groups must be
large enough and preferably taken together in a multi-centre study.
http://lymediseaseguide.org/lyme-disease-ms-bacteria-cause
http://www.ncbi.nlm.nih.gov/pubmed/15617845
