Bartonella — a stealth, vascular, intracellular infection — can directly impact the skin.

 

 

Jessica Nohren thanks for allowing me to share 

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CYSTIC ACNE
what if it’s not just “hormones”?!

We are so quick to blame breakouts on puberty, stress, or diet… but deep, painful, cystic acne can be a sign of something much deeper going on inside the body.

#Bartonella — a stealth, vascular, intracellular infection — can directly impact the skin.

Here’s what’s happening:
Bartonella infects the lining of blood vessels (endothelium)
Triggers chronic inflammation + immune dysregulation
Disrupts normal skin healing and oil gland function
Promotes formation of painful, deep cystic lesions
Can cause streaks, rashes, and persistent “unexplained” skin issues

This isn’t surface-level acne…
This is systemic inflammation showing up through your skin.

And here’s the part no one is talking about

Testing for Bartonella is VERY limited
Blood tests often come back negative even when infection is present
Multiple species (henselae, quintana, etc.) are rarely all tested
Bartonella lives inside cells, making it hard to detect in blood
Diagnosis is often clinical - based on symptoms--not labs (by and ILADS trained Physician)

So people get told:
“It’s just #acne.”
“It’s hormonal.”
“Try another cream.”

Meanwhile… the root cause is never addressed.

If acne is:
Deep, cystic, and painful
Resistant to typical treatments
Paired with fatigue, anxiety, nerve pain, rage, stretch-mark-like rashes, or otherBartonella symptoms

It’s time to look deeper.

Because sometimes…
Your #skin is telling the story your labs are missing....

#acnetreatment #acne #acneproneskin #acnecare #acnescars #acnescartreatment #skincare #skincaretips #skinhealth #skincareroutine #SkincareEssentials #cysticacne #cysticacnetreatment #FaceToFace #bartonella #RootCauseHealing #rootcause #lymedisease #lymediseaseawareness #Lyme #pimple #pimplepoppe

 

 

 

 

Chris Masterjohn and Dr Datis Kharrazian have been talking about importance of Vitamin A for “immune tolerance” for years, particularly regarding

Chris Masterjohn and Dr Datis Kharrazian have been talking about importance of Vitamin A for “immune tolerance” for years, particularly regarding
immune tolerance,
preventing autoimmunity, and managing allergies !!

Masterjohn has focused on the synergistic relationship between Vitamin A and Vitamin D, he argues they work together to prevent Autoimmunity.

Deficiencies in Vitamins A and D can lead to post-infectious autoimmunity by failing to properly regulate the immune system's response !

Masterjohn notes that Vitamin A deficiency is a significant, often overlooked cause of allergies and can lead to increased reactivity (e.g., in the gut or eyes).

Vitamin A helps suppress overactive Th17 cells while boosting immunity against infections.

He advises caution with high-dose supplementation, often recommending testing blood retinol levels (aiming for 600-700 µg/L) and

using liver (within safe, moderate amounts) as a primary source.
( The Weston A. Price Foundation )

Dr Kharrazian says Vitamin A is a key
modulator required to boost anti-inflammatory regulatory T cells (T-reg cells).

He uses Vitamin A help the immune system stop reacting to food proteins and environmental triggers (improving "oral tolerance").

He has argued that addressing micronutrient deficiencies like Vitamin A is crucial, especially when dealing with chemical sensitivities and
intestinal barrier issues.

Using Vitamin A as a modulator, not just an immune booster, to restore balance.
Preventing the loss of tolerance that leads to autoimmune diseases.

Evidence supports the role of Vitamin A in strengthening the gastrointestinal (GI) barrier and regulating immune responses, although the ability to specifically "stop" reactions to salicylates and tannins is not yet a direct clinical claim.

Effect on GI Permeability
Vitamin A (and its metabolite, retinoic acid) is essential for maintaining the physical barrier of the gut.

Tight Junctions
VItamin A directly improves the expression of "gatekeeper" proteins like occludin, claudin-1, and ZO-1, which seal the gaps between intestinal cells.

VItamin A It promotes the secretion of mucin !! (MUC2), the protective gel layer that prevents pathogens and irritating chemicals like tannins from reaching the sensitive gut lining !!

Repair:
Supplementation has been shown to reduce markers of "leaky gut" (such as serum zonulin and diamine oxidase) following infection-induced damage.

Interaction with Salicylates and Tannins
While Vitamin A doesn't neutralize these chemicals directly, it addresses the consequences of sensitivity:
Tannins can irritate the gut lining
Vitamin A helps by reinforcing the mucus layer that acts as a buffer against these astringent compounds.

Salicylates Intolerance often involves mast cell overactivity and a lack of immune tolerance.

Vitamin A is crucial for inducing Regulatory T cells (Tregs), which are the primary cells responsible for teaching the immune system not to overreact to food proteins and chemicals.

Reducing Antibodies and Food Reactivity
There is no clinical evidence that taking Vitamin A will immediately "delete" existin antibodies, but it modulates the environment where they act !!

Immune Tolerance:

By promoting Treg production, Vitamin A helps the body transition from an "attack" mode (autoimmunity/reactivity) to a "tolerance" mode.

It has been shown to reduce pro-inflammatory cytokines (like IL-6 and TNF-α) which are often elevated when food sensitivities trigger joint pain or systemic flares.


Anti-Inflammatory Effect
In post-infection states, Vitamin A can reduce the recruitment of inflammatory cells to the gut, potentially calming the "false alarms" that occur when your friend eats reactive foods.



Note on Safety: Vitamin A is fat-soluble and can be toxic in high doses. It is critical to consult a professional to check blood levels before starting high-dose supplementation, especially if already dealing with a stressed liver or metabolic system post-infection.

Vitamin A and vitamin D regulate the microbial complexity, barrier function and the mucosal

While Vitamin A is not a direct antidote for salicylate or tannin reactions, it plays a critical role in supporting the systems that prevent these symptoms 

How Vitamin A Addresses Respiratory "Air Hunger"

Vitamin A (retinoic acid) is essential for maintaining the integrity of the respiratory epithelium—the lining of the airways.

Reduces Airway Hyperresponsiveness: Vitamin A deficiency is linked to "twitchy" airways that overreact to triggers.

Supplementation may help stabilize these tissues, making them less likely to spasm or feel "tight" when your friend is exposed to salicylates.

Alveolar Support: Vitamin A is vital for the formation and maintenance of lung alveoli, the tiny sacs where oxygen exchange happens.
Strengthening these can theoretically improve the efficiency of breathing

Stopping GI Permeability (The "Root" of the Reaction)
 Vitamin A is a primary regulator of intestinal tight junctions.

By repairing the "leaky gut", Vitamin A can reduce the amount of undigested salicylates and tannins that enter the bloodstream.

It promotes T-regulatory (Treg) cells, which signal the immune system to stop attacking harmless food chemicals. This can help lower the circulating antibodies and reduce the systemic "alarm" that leads to joint pain and air hunger.

Why Salicylates Cause Air Hunger
In people with this intolerance, salicylates interfere with the COX enzyme pathway, leading to an overproduction of leukotrienes.

Leukotrienes are powerful chemicals that cause bronchoconstriction (narrowing of airways) and mucus buildup, which directly causes the sensation of air hunger.

Salicylates can also act as a direct respiratory stimulant in the brain's medulla, which can trick the body into hyperventilating even when oxygen levels are normal.

Important Precautions
Dosage is Critical: While Vitamin A helps, excessive amounts can actually worsen asthma-like symptoms and cause toxicity.