Atteinte de la borréliose chronique persistante , babesia et bartonella ; le déni est grand , battons nous pour la faire connaitre la borréliose de lyme et ses coinfections ; 8 ans de bi et tri therapies d'antibiotiques auxquels j'ai ajouté une partie du protocole Buhner et autres http://francelyme.fr
Precision Diagnosis for Tick-Borne Diseases?
directorsblog.nih.gov
For many of us who enjoy roaming the great outdoors, there are some…
A Brief Overview of Lyme Bacteria Behavior
A Brief Overview of
Lyme Bacteria Behavior
Lyme Disease is a complex systemic disease that is caused by highly motile bacterium in the spirochete family. The bacteria Borrelia burgdorferi was first isolated from the skin of a Lyme patient with the distinctive bull’s-eye rash in the early 1980s. Since that time, culturing the elusive bacterium has been difficult, frustratingly unpredictable, and miserably inconsistent.
Borrelia burgdorferi can very quickly move from the site of a tick bite directly into the circulatory system where it can circulate throughout the entire body. Most bacterial infections that enter the blood stream are trapped within the blood vessels and consequently are easier to kill with antibiotics than bacteria that are dispersed throughout many tissues and organs.
Borrelia burgdorferi is a unique bacterium that has a distinct and insidious method for passing through the capillary walls of human blood vessels and making its way into whatever tissue that waits on the other side of the capillary wall.
If the spirochete finds a tissue it likes and a place where it can thrive, the bacteria will survive and multiply. If conditions are unfavorable the bacteria may die, but more disturbing than the bacteria’s ability to migrate and penetrate blood vessels is recent evidence from the NIH Rocky Mountain Labs that this particular species of spirochete like many of its cousins may have a protective cyst-like form.
When stressed the Lyme bacteria under certain conditions will shed a portion of its cell wall and extrude a membrane wrapped vesicle that is a spherical shaped blob.
These blebs appear to be minimally metabolically active which means they can essentially lay dormant and resist antibiotics. They stimulate the immune system and contain the bacteria’s DNA. In some species of spirochetes blebs have been observed to harbor classical-form spirochetes inside them.
The cyst-like L-form or bleb may become metabolically active at a more favorable time and then go on to produce fully functional classical-form spirochetes that can once again roam the human body in a motile form.
The Lyme bacteria begin their passage through blood vessels by attaching themselves to the endothelial cells lining the inside of the blood vessel. Then it continually twitches and pushes itself as deep as it can go.
The simple act of attaching to the endothelial cells lining the blood vessels initiates the endothelial cells to release digestive enzymes including tissue plasminogen, proteases, and basement membrane laminase. This starts a cascade of cellular degradation that ultimately leads to leaky gaps between the cells that make up the blood vessel walls.
Once the bacteria are out of the blood stream they are able to travel deep inside many organs and tissues of the human body. In animal models, the Lyme spirochete within mere hours of a tick bite, causes a dramatic breakdown of the blood-brain-barrier. This breakdown of the BBB has been observed in rats, mice, hamsters, dogs, cats, and monkeys.
Why is it important to understand this mechanism of blood vessel penetration when we are discussing Lyme disease diagnostic tests?
The fact that the Lyme bacteria can escape the blood stream very early in the infection is fundamental to understanding why the Lyme tests are so fallible and why there may never be a Lyme test that is truly trustworthy.
Just because you kill all the cockroaches that you see in your basement doesn’t mean there aren’t some in hidden in the walls and cupboards!
(The blood vessels in the brain usually prevent bacteria and toxins from entering the brain. A loss of this barrier not only allows the Lyme bacteria to enter the brain but also other undesirable substances such as inflammatory cells, toxins and other bacteria enter the brain. Creating a perforated BBB opens the patient up to further complications!)
It is the bacteria’s ability to exit the blood stream, hide, and survive that makes Lyme disease so difficult to detect with any one test. It is the unique microbiology of this bacterium that gives it the ability to hide and survive undetected within the human body.
The uniqueness of this bacterium is why using other bacterial diseases as a model for Lyme disease is not only inaccurate but it leads to misunderstandings within the medical community on how to properly diagnose and treat Lyme disease.
For years the Lyme pathogen has been treated as though it were like other bacteria that are obligated to stay within the bloodstream, but Borrelia burgdorferi does not play by the same rules as other bacterial infections!
When physicians try to simplify their understanding of this disease by treating it like other bacterial infections, they disregard the uniqueness of these bacteria to hide, thrive and survive.
If a doctor uses a “one-size-fits-all” approach to diagnosis and treatment, the result will be one population of patients that never get diagnosed, and another population of patients that will relapse from incomplete treatment.
It is not a failure of medical science that allows relapses to occur. It is a lack of understanding medical science that allows patients with Lyme disease to suffer needlessly.
Infections caused by the family of bacteria known as Borrelia are unique in their microbiology and cannot be dismissed with a rubber stamped one-treatment-fits-all approach. Lyme disease is not a sore throat, it is not Syphilis, and it is not Tuberculosis. It is Borreliosis: A very misunderstood collection of diseases cause by the Borrelia group of bacteria.
The Borrelia genus of bacteria represent over 40 disease causing bacteria that are all related to each other and all share the common gift of being highly variable and adaptive.
Lyme disease is not a single disease, it is part of a growing family of pathogens that basically cause variations of Relapsing Fever. These similar Lyme-like bacteria should really be considered collectively as one disease. Lyme disease is part of the bigger picture that we can refer to as Borreliosis.
Lyme Bacteria Behavior
Lyme Disease is a complex systemic disease that is caused by highly motile bacterium in the spirochete family. The bacteria Borrelia burgdorferi was first isolated from the skin of a Lyme patient with the distinctive bull’s-eye rash in the early 1980s. Since that time, culturing the elusive bacterium has been difficult, frustratingly unpredictable, and miserably inconsistent.
Borrelia burgdorferi can very quickly move from the site of a tick bite directly into the circulatory system where it can circulate throughout the entire body. Most bacterial infections that enter the blood stream are trapped within the blood vessels and consequently are easier to kill with antibiotics than bacteria that are dispersed throughout many tissues and organs.
Borrelia burgdorferi is a unique bacterium that has a distinct and insidious method for passing through the capillary walls of human blood vessels and making its way into whatever tissue that waits on the other side of the capillary wall.
If the spirochete finds a tissue it likes and a place where it can thrive, the bacteria will survive and multiply. If conditions are unfavorable the bacteria may die, but more disturbing than the bacteria’s ability to migrate and penetrate blood vessels is recent evidence from the NIH Rocky Mountain Labs that this particular species of spirochete like many of its cousins may have a protective cyst-like form.
When stressed the Lyme bacteria under certain conditions will shed a portion of its cell wall and extrude a membrane wrapped vesicle that is a spherical shaped blob.
These blebs appear to be minimally metabolically active which means they can essentially lay dormant and resist antibiotics. They stimulate the immune system and contain the bacteria’s DNA. In some species of spirochetes blebs have been observed to harbor classical-form spirochetes inside them.
The cyst-like L-form or bleb may become metabolically active at a more favorable time and then go on to produce fully functional classical-form spirochetes that can once again roam the human body in a motile form.
The Lyme bacteria begin their passage through blood vessels by attaching themselves to the endothelial cells lining the inside of the blood vessel. Then it continually twitches and pushes itself as deep as it can go.
The simple act of attaching to the endothelial cells lining the blood vessels initiates the endothelial cells to release digestive enzymes including tissue plasminogen, proteases, and basement membrane laminase. This starts a cascade of cellular degradation that ultimately leads to leaky gaps between the cells that make up the blood vessel walls.
Once the bacteria are out of the blood stream they are able to travel deep inside many organs and tissues of the human body. In animal models, the Lyme spirochete within mere hours of a tick bite, causes a dramatic breakdown of the blood-brain-barrier. This breakdown of the BBB has been observed in rats, mice, hamsters, dogs, cats, and monkeys.
Why is it important to understand this mechanism of blood vessel penetration when we are discussing Lyme disease diagnostic tests?
The fact that the Lyme bacteria can escape the blood stream very early in the infection is fundamental to understanding why the Lyme tests are so fallible and why there may never be a Lyme test that is truly trustworthy.
Just because you kill all the cockroaches that you see in your basement doesn’t mean there aren’t some in hidden in the walls and cupboards!
(The blood vessels in the brain usually prevent bacteria and toxins from entering the brain. A loss of this barrier not only allows the Lyme bacteria to enter the brain but also other undesirable substances such as inflammatory cells, toxins and other bacteria enter the brain. Creating a perforated BBB opens the patient up to further complications!)
It is the bacteria’s ability to exit the blood stream, hide, and survive that makes Lyme disease so difficult to detect with any one test. It is the unique microbiology of this bacterium that gives it the ability to hide and survive undetected within the human body.
The uniqueness of this bacterium is why using other bacterial diseases as a model for Lyme disease is not only inaccurate but it leads to misunderstandings within the medical community on how to properly diagnose and treat Lyme disease.
For years the Lyme pathogen has been treated as though it were like other bacteria that are obligated to stay within the bloodstream, but Borrelia burgdorferi does not play by the same rules as other bacterial infections!
When physicians try to simplify their understanding of this disease by treating it like other bacterial infections, they disregard the uniqueness of these bacteria to hide, thrive and survive.
If a doctor uses a “one-size-fits-all” approach to diagnosis and treatment, the result will be one population of patients that never get diagnosed, and another population of patients that will relapse from incomplete treatment.
It is not a failure of medical science that allows relapses to occur. It is a lack of understanding medical science that allows patients with Lyme disease to suffer needlessly.
Infections caused by the family of bacteria known as Borrelia are unique in their microbiology and cannot be dismissed with a rubber stamped one-treatment-fits-all approach. Lyme disease is not a sore throat, it is not Syphilis, and it is not Tuberculosis. It is Borreliosis: A very misunderstood collection of diseases cause by the Borrelia group of bacteria.
The Borrelia genus of bacteria represent over 40 disease causing bacteria that are all related to each other and all share the common gift of being highly variable and adaptive.
Lyme disease is not a single disease, it is part of a growing family of pathogens that basically cause variations of Relapsing Fever. These similar Lyme-like bacteria should really be considered collectively as one disease. Lyme disease is part of the bigger picture that we can refer to as Borreliosis.
L' Abri des Possibles d'Auch organisait ce dimanche 24 juin une fin de journée consacrée à la Maladie de Lyme
MALADIE DE LYME
L' Abri des Possibles d'Auch organisait ce dimanche 24 juin une fin de journée consacrée à la Maladie de Lyme (reconnue pour la première fois en 1975 dans cette ville, Lyme, du Connecticut aux USA). Plus de 100 personnes avaient fait le déplacement pour s'informer et s'exprimer sur cette maladie qui n'est plus une maladie rare, car elle gagne du terrain mais reste mal connue.
En première partie, le Dr Thierry Medynski, généraliste, bon connaisseur de la maladie, a exposé de façon très documentée comment cet acarien, une tique, injecte de très nombreuses bactéries dans le système sanguin de la personne piquée, ce qui est à l'origine d'une infection, la borréliose, qui peut avoir des conséquences gravissimes (paralysie faciale, atteintes cardiaques, oculaires, neuropathie). Nombreuses maladies chroniques sans causes apparentes peuvent être consécutives à une piqûre de tique. Le monde de la médecine est partagé sur la marche à suivre quant au diagnostic et au traitement.
Au cours du débat, de nombreuses personnes ont pu obtenir des précisions ou apporter leur témoignage. Puis un film de Chantal Perrin, "Quand les tiques attaquent", déroulait le sujet à partir de trois personnes atteintes de la maladie, militant pour qu'elle soit mieux connue.
Cette programmation tombait à point nommé au moment où le gouvernement rendait enfin public (le 20 juin) un Protocole national de diagnostic et de soins (PNDS) organisant la thérapeutique de cette maladie. Des personnes de l'Abri des Possibles se sont fortement mobilisées pour rendre cette manifestation… possible : qu'elles en soient vivement remerciées. Leur objectif est de faire fonctionner dans le Gers une antenne locale de l'Association nationale France Lyme. Yves Faucoup [texte et photos]
. L'Abri des Possibles (café associatif), Place Barbès, 32000 Auch.
. http://blog.abridespossibles.fr/
Lyme : des nouvelles recos mais pas la fin des polémiques
francais.medscape.com
Les nouvelles recommandations sur la borréliose de Lyme et autres…
For the first time, a committee of the US Department of Health and Human Services has recognized Bartonella as a tick-borne disease.
For the first time, a committee of the US Department of Health and
Human Services has recognized Bartonella as a tick-borne disease.
The committee members voted unanimously to encourage to improve testing and increased research on epidemiology and treatment.
The committee members voted unanimously to encourage to improve testing and increased research on epidemiology and treatment.
The committee members voted unanimously to encourage to improve testing and increased research on epidemiology and treatment.
———-
Report of the Other Tick-Borne Diseases and Coinfections Subcommittee to the Tick-Borne Disease Working Group
Background: Why other tick-borne diseases and co-infections are important
Co-infection of ticks with human and animal pathogens is more widespread than is commonly recognized by both medical professionals and the public. Ticks contain multiple tick-borne pathogens, which can be transmitted with a single tick bite. Some tick-borne pathogens can be transmitted within a short period of time after a tick bite (i.e., Borrelia hermsii and Powassan virus), although other tick-borne pathogens such as Anaplasma, Ehrlichia, Babesia,Rickettsia and other borrelia species take longer. Transmission for some tick-borne diseases is also possible by blood transfusion (Babesia, Anaplasma, Bartonella, tick-borne relapsing fever), solid organ transplantation (Babesia), and through maternal-fetal transmission (Babesia, Bartonella, relapsing fever borreliae, certain arthropod borne flaviviruses). Increased awareness with appropriate testing and treatment is therefore important to help decrease morbidity and mortality from other tick-borne diseases and co-infections.
Current microbiologic science suggests that some tick-borne pathogens and co-infections can infect humans both acutely and chronically. These pathogens can persist in an indolent manner and reactivate periodically causing relapsing and remitting illness. Clinical observation of health care providers who see tick-borne disease patients observe that in persistently symptomatic patients more than one tick-borne disease is often present. It should be kept in mind that these co-infections may have been acquired sequentially, not necessarily with one single tick bite. Peer reviewed literature suggest coinfection of a patient with Babesia and Borrelia or with Ehrlichia, Anaplasma, and Borrelia result in increased symptoms and a longer duration of illness. The burden of borreliosis alone is documented, and society suffers in terms of lost productivity and patients requiring more care and support. Given that preliminary peer-reviewed data and clinical expert opinion notes increased morbidity when a patient has more than one tick-borne infection, the burden of coinfection is likely higher than that of borreliosis alone. Identifying the incidence of coinfection among chronically ill patients, while establishing effective diagnostic and treatment regimens, will help decrease the burden of illness and associated suffering.
https://www.hhs.gov/ash/advisory-committees/tickbornedisease/reports/other-tbds-2018-5-9/index.html#.Wvp355hLKlc.facebook
Report of the Other Tick-Borne Diseases and Coinfections Subcommittee to the Tick-Borne Disease Working Group
Background: Why other tick-borne diseases and co-infections are important
Co-infection of ticks with human and animal pathogens is more widespread than is commonly recognized by both medical professionals and the public. Ticks contain multiple tick-borne pathogens, which can be transmitted with a single tick bite. Some tick-borne pathogens can be transmitted within a short period of time after a tick bite (i.e., Borrelia hermsii and Powassan virus), although other tick-borne pathogens such as Anaplasma, Ehrlichia, Babesia,Rickettsia and other borrelia species take longer. Transmission for some tick-borne diseases is also possible by blood transfusion (Babesia, Anaplasma, Bartonella, tick-borne relapsing fever), solid organ transplantation (Babesia), and through maternal-fetal transmission (Babesia, Bartonella, relapsing fever borreliae, certain arthropod borne flaviviruses). Increased awareness with appropriate testing and treatment is therefore important to help decrease morbidity and mortality from other tick-borne diseases and co-infections.
Current microbiologic science suggests that some tick-borne pathogens and co-infections can infect humans both acutely and chronically. These pathogens can persist in an indolent manner and reactivate periodically causing relapsing and remitting illness. Clinical observation of health care providers who see tick-borne disease patients observe that in persistently symptomatic patients more than one tick-borne disease is often present. It should be kept in mind that these co-infections may have been acquired sequentially, not necessarily with one single tick bite. Peer reviewed literature suggest coinfection of a patient with Babesia and Borrelia or with Ehrlichia, Anaplasma, and Borrelia result in increased symptoms and a longer duration of illness. The burden of borreliosis alone is documented, and society suffers in terms of lost productivity and patients requiring more care and support. Given that preliminary peer-reviewed data and clinical expert opinion notes increased morbidity when a patient has more than one tick-borne infection, the burden of coinfection is likely higher than that of borreliosis alone. Identifying the incidence of coinfection among chronically ill patients, while establishing effective diagnostic and treatment regimens, will help decrease the burden of illness and associated suffering.
https://www.hhs.gov/ash/advisory-committees/tickbornedisease/reports/other-tbds-2018-5-9/index.html#.Wvp355hLKlc.facebook
hhs.gov
Report of the Other TBDS and Co-Infections Subcommittee to the TBDWG to consider for the Report to Congress
———-
Report of the Other Tick-Borne Diseases and Coinfections Subcommittee to the Tick-Borne Disease Working Group
Background: Why other tick-borne diseases and co-infections are important
Co-infection of ticks with human and animal pathogens is more widespread than is commonly recognized by both medical professionals and the public. Ticks contain multiple tick-borne pathogens, which can be transmitted with a single tick bite. Some tick-borne pathogens can be transmitted within a short period of time after a tick bite (i.e., Borrelia hermsii and Powassan virus), although other tick-borne pathogens such as Anaplasma, Ehrlichia, Babesia,Rickettsia and other borrelia species take longer. Transmission for some tick-borne diseases is also possible by blood transfusion (Babesia, Anaplasma, Bartonella, tick-borne relapsing fever), solid organ transplantation (Babesia), and through maternal-fetal transmission (Babesia, Bartonella, relapsing fever borreliae, certain arthropod borne flaviviruses). Increased awareness with appropriate testing and treatment is therefore important to help decrease morbidity and mortality from other tick-borne diseases and co-infections.
Current microbiologic science suggests that some tick-borne pathogens and co-infections can infect humans both acutely and chronically. These pathogens can persist in an indolent manner and reactivate periodically causing relapsing and remitting illness. Clinical observation of health care providers who see tick-borne disease patients observe that in persistently symptomatic patients more than one tick-borne disease is often present. It should be kept in mind that these co-infections may have been acquired sequentially, not necessarily with one single tick bite. Peer reviewed literature suggest coinfection of a patient with Babesia and Borrelia or with Ehrlichia, Anaplasma, and Borrelia result in increased symptoms and a longer duration of illness. The burden of borreliosis alone is documented, and society suffers in terms of lost productivity and patients requiring more care and support. Given that preliminary peer-reviewed data and clinical expert opinion notes increased morbidity when a patient has more than one tick-borne infection, the burden of coinfection is likely higher than that of borreliosis alone. Identifying the incidence of coinfection among chronically ill patients, while establishing effective diagnostic and treatment regimens, will help decrease the burden of illness and associated suffering.
https://www.hhs.gov/…/repor…/other-tbds-2018-5-9/index.html…
Report of the Other Tick-Borne Diseases and Coinfections Subcommittee to the Tick-Borne Disease Working Group
Background: Why other tick-borne diseases and co-infections are important
Co-infection of ticks with human and animal pathogens is more widespread than is commonly recognized by both medical professionals and the public. Ticks contain multiple tick-borne pathogens, which can be transmitted with a single tick bite. Some tick-borne pathogens can be transmitted within a short period of time after a tick bite (i.e., Borrelia hermsii and Powassan virus), although other tick-borne pathogens such as Anaplasma, Ehrlichia, Babesia,Rickettsia and other borrelia species take longer. Transmission for some tick-borne diseases is also possible by blood transfusion (Babesia, Anaplasma, Bartonella, tick-borne relapsing fever), solid organ transplantation (Babesia), and through maternal-fetal transmission (Babesia, Bartonella, relapsing fever borreliae, certain arthropod borne flaviviruses). Increased awareness with appropriate testing and treatment is therefore important to help decrease morbidity and mortality from other tick-borne diseases and co-infections.
Current microbiologic science suggests that some tick-borne pathogens and co-infections can infect humans both acutely and chronically. These pathogens can persist in an indolent manner and reactivate periodically causing relapsing and remitting illness. Clinical observation of health care providers who see tick-borne disease patients observe that in persistently symptomatic patients more than one tick-borne disease is often present. It should be kept in mind that these co-infections may have been acquired sequentially, not necessarily with one single tick bite. Peer reviewed literature suggest coinfection of a patient with Babesia and Borrelia or with Ehrlichia, Anaplasma, and Borrelia result in increased symptoms and a longer duration of illness. The burden of borreliosis alone is documented, and society suffers in terms of lost productivity and patients requiring more care and support. Given that preliminary peer-reviewed data and clinical expert opinion notes increased morbidity when a patient has more than one tick-borne infection, the burden of coinfection is likely higher than that of borreliosis alone. Identifying the incidence of coinfection among chronically ill patients, while establishing effective diagnostic and treatment regimens, will help decrease the burden of illness and associated suffering.
https://www.hhs.gov/…/repor…/other-tbds-2018-5-9/index.html…
hhs.gov
Report of the Other TBDS and Co-Infections Subcommittee to the TBDWG to consider for the Report to Congress
Bartonella: More Than Skin Deep
Many doctors and patients have reported an association between skin striae and #Bartonella infection. To date, an association has only been documented in a single
published case report. An award-winning researcher at the University
of Minnesota, Dr. Marna Ericson, is looking into the possible link
between Bartonella infection and skin striae as well as potential
involvement in Gulf War Illness and more. Galaxy Diagnostics is a
research partner on these projects. To learn more and to support this
important Bartonella research, see here:
crowdfund.umn.edu
Double
Your Impact! Make a gift before January 1, 2018, and it will be matched
dollar-for-dollar up to $10,000! Special thanks to Steve Sim and his
wife Marilyn Einstein and Nancy and Mike Goodman who are generously
supporting Dr. Marna Ericson's research by matching up to $10,000 raised
for this fu...
Vanessa Paradis : Confrontée à une maladie mortelle !
https://www.francedimanche.fr/actualites/vanessa-paradis-confrontee-a-une-maladie-mortelle
En effet, en cas de morsure, il faut avant toute chose arracher la tique, de façon à ne pas laisser sa tête sous la peau, puis procéder à une sérieuse désinfection de la piqûre.
non on n'arrache pas la tique mais on enlève avec un tire tique , quelles mauvaises informations
Jyotsna Shah, PhD from Igenex. “Babesiosis a World Wide Problem”, at the ILADS conference in Warsaw.
Jyotsna Shah, PhD from Igenex. “Babesiosis a World Wide Problem”, at the ILADS conference in Warsaw.
Christian Perronne, MD, PhD, at the ILADS conference in Warsaw. Epidemiology of Lyme borreliosis in Europe.
Christian Perronne, MD, PhD, at the ILADS conference in Warsaw.
Epidemiology of Lyme borreliosis in Europe.
Saxony Germany, 70,5% infected Ticks with Rickettsia
Governor Cuomo Announces New Public-Private Research Collaboration to Advance Diagnosis and Treatment of…
54 million dollars pledged to develop diagnostics and therapies for tick-borne disease.
Yes, we have duly noted the below line and some of the careful and questionable wording in this article:
Yes, we have duly noted the below line and some of the careful and questionable wording in this article:
“When diagnosed correctly and treated, most Lyme disease patients recover within two weeks.”
Still, we are glad Governor Cuomo understands the desperate need of TBD sufferers and is taking action to help.
https://www.governor.ny.gov/…/governor-cuomo-announces-new-…
Still, we are glad Governor Cuomo understands the desperate need of TBD sufferers and is taking action to help.
https://www.governor.ny.gov/…/governor-cuomo-announces-new-…
1. The Lyme vaccine! 2. Does Babesia cause a herxheimer? 3. The confusing overlap between Babesia and Bartonella
-12:16
Dr. Steven Phillips & Dana Parish
Because you asked...
Our brand new video is all about:
1. The Lyme vaccine!
2. Does Babesia cause a herxheimer?
3. The confusing overlap between Babesia and Bartonella.
Our brand new video is all about:
1. The Lyme vaccine!
2. Does Babesia cause a herxheimer?
3. The confusing overlap between Babesia and Bartonella.
We SO appreciate your ideas, suggestions, and questions and will
continue to do our best to provide the information you’re looking for.
Thanks for being here!
If you find this video helpful, please like and share! #TheTruthAboutLyme
Thanks for being here!
If you find this video helpful, please like and share! #TheTruthAboutLyme
Precision Diagnosis for Tick-Borne Diseases?
directorsblog.nih.gov
For many of us who enjoy roaming the great outdoors, there are some…
VIRAS response to the British Medical Journal report on "Obstacles to diagnosis and treatment of Lyme disease in the USA: a qualitative study"
VIRAS
response to the British Medical Journal report on "Obstacles to
diagnosis and treatment of Lyme disease in the USA: a qualitative study"
http://bmjopen.bmj.com/content/8/6/e021367.responses
bmjopen.bmj.com
Objective For many individuals with Lyme disease, prompt treatment leads to rapid…
Tertiary Lyme Disease
Tertiary
stage Lyme is very similar to tertiary stage Syphilis, and causes
exactly the same changes in the brain as seen in Alzheimer's disease.
Great to read this research from Drexel university.
omicsonline.org
Penny Lane on Her Morgellons Disease Doc, The Pain of Others
Filmmaker
Penny Lane has made a documentary about Morgellons Disease called "The
Pain of Others", which approaches the subject matter from a totally
different perspective than our film
"Skin Deep: The Battle Over Morgellons." But Ms. Lane has spoken up in
her latest interview in Filmmaker Magazine: "The mainstream says
there’s nothing physiological happening and that it is all a delusion,
but the mainstream is often wrong. I won’t pretend to take the blatant
pseudoscience or conspiracy theories seriously; that is not required of
me intellectually or ethically. But I do believe there is a real
possibility that there exists a skin condition producing the fibers that
are the key diagnostic of Morgellons. A small number of scientists are
investigating this very plausible theory, despite the scorn heaped upon
them by the mainstream. In the meantime, a lot of people whose suffering
and pain is not currently understood by their doctors to be “real” are
drawn toward the clarity of a Morgellons (or chronic Lyme, or ME, or
MCS) diagnosis. As Anne Carson said (I think), 'One of the principle
qualities of pain is that it demands an explanation.'"
Recent-onset dilated cardiomyopathy associated with Borrelia burgdorferi infection. -
ncbi.nlm.nih.gov
Herz. 2015 Sep;40(6):892-7. doi: 10.1007/s00059-015-4308-1. Epub 2015 May 5.…
A doctor said her little-known disorder was all in her head
Survey
reports more than 75% of POTS patients were initially told their
symptoms were psychological. 25% were treated for a mental disorder
before they got an accurate diagnosis.
UK study findings are similar. Out of 779 UK patients, 48% were told they had a psychiatric disorder or were hypochondriacs.
UK study findings are similar. Out of 779 UK patients, 48% were told they had a psychiatric disorder or were hypochondriacs.
cnn.com
Super Bowl MVP Nick Foles' wife Tori has a little-known debilitating…
Tiques : 10 répulsifs sur 13 jugés efficaces par l'UFC-Que-Choisir
"Parmi
ces produits efficaces :Insect écran familles, Mousti fluid zones
tropicales et à risques, Cinq sur cinq actif d’origine végétale,
Mosi-guard natural lotion anti insectes, Cinq sur cinq spray UV, Apaisyl
lait haute protection ou encore Puressentiel antipique bébé."
Maladie de lyme : la prévention avant tout
france3-regions.francetvinfo.fr
Le Limousin fait partie des trois régions où la maladie de lyme, transmise…
Nematodes, strongyloides, and amoebas
Nematodes,
strongyloides, and amoebas*
Nematodes, strongyloides, and amoebas, all like moist mud. Amoebas can enter the body including the brain through swimming in contaminated warm water swimming holes, hot mud baths and enter through mucous membranes or ruptured ear drums, Stongyloides most likely through open sores and ingestion, nematodes might enter through unwashed vegetables, and open sores, but mud and dirt are most likely to harbor these parasites.
Nematodes, strongyloides, and amoebas, all like moist mud. Amoebas can enter the body including the brain through swimming in contaminated warm water swimming holes, hot mud baths and enter through mucous membranes or ruptured ear drums, Stongyloides most likely through open sores and ingestion, nematodes might enter through unwashed vegetables, and open sores, but mud and dirt are most likely to harbor these parasites.
These are images from an unpublished case of an 11 year old girl with "MS". Unfortunately her condition was allowed to progress to the point where she was restricted to a wheelchair.
These
are images from an unpublished case of an 11 year old girl with "MS".
Unfortunately her condition was allowed to progress to the point where
she was restricted to a wheelchair.
There was no need for a brain biopsy or spinal fluid because her blood was loaded with three pathogens.
Completely invisible with normal blood stains and invisible with normal light microscopy, this girl's blood using species specific DNA FISH stains showed the presence of Borrelia burgdorferi, Borrelia myamotoi, and nematode parasites. (Using Cytochrome-5 stain) and actin-green stain.
There was no need for a brain biopsy or spinal fluid because her blood was loaded with three pathogens.
Completely invisible with normal blood stains and invisible with normal light microscopy, this girl's blood using species specific DNA FISH stains showed the presence of Borrelia burgdorferi, Borrelia myamotoi, and nematode parasites. (Using Cytochrome-5 stain) and actin-green stain.
Using Actin stain the outline of nematode eggs could be seen.
Using FISH stain for Borrelia flagellin B a round body is clearly seen. The role of round bodies, L-Forma, and granular forms is unclear and may play a role in alternative reproductive methods or to increase survival rate when attacked by host immune systems?
Using FISH stain for Borrelia flagellin B a round body is clearly seen. The role of round bodies, L-Forma, and granular forms is unclear and may play a role in alternative reproductive methods or to increase survival rate when attacked by host immune systems?
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