"These results suggest that spirochetal residues left after bacterial demise, due to treatment or otherwise, may continue to be pathogenic to the central nervous system"

"These results suggest that spirochetal residues left after bacterial demise, due to treatment or otherwise, may continue to be pathogenic to the central nervous system"

That's interesting in itself : dead bugs or even bits of bugs cause an inflammatory response in the CNS. BUT are they REALLY non-viable residues? Or might they in fact be dormant forms, cysts, granules etc. that may RESURRECT and MULTIPLY?

Non-viable Borrelia burgdorferi induce inflamm... [Neurosci Lett. 2013] - PubMed - NCBI

www.ncbi.nlm.nih.gov

In this study we assessed the ability of bacteria (heat killed or sonicated) to inflammatory mediators and cell death.