collecte section Bourgogne

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Spiroplasma - Creutzfeldt-Jakob disease in Australia

<Look familiar? Sounds al ot like spirochete + mycoplasma. These Spiroplasma travel in the same spiral drilling motion as spirochetes however hide their protein (identifying markers) from immune system etc by essentially turning inside out. Lyme Borrelia has been shown to turn itself inside out and form spheroplast or cystic form in a hostile environment - immune or antibiotics etc.>>

Interesting...






Wednesday, September 16, 2009


Spiroplasma - Creutzfeldt-Jakob disease in Australia

Look familiar? Sounds alot like spirochete + mycoplasma. TheseSpiroplasma travel in the same spiral drilling motion as spirochetes however hide their protein (identifing markers) from immune system etc by essentially turning inside out. Lyme Borellia has been shown to turn itself inside out and form spheroplast or cystic form in a hostile environment - immune or antibiotics etc.

These invasive pathogens are so dangerous that surgical equipment standard sterilization doesn't work so notification must be provided. They have not proved blood transfusion transmission yet...though infection has been confirmed from organ transplant studies.

Creutzfeldt-Jakob disease (looks like evolved lyme to me...and it comes from arthropod (incl ticks), insects and vertebrates, and seems to have similar vector life cycle patterns from what I gather. The 2009 Australian Health Report has implemented a strategy to help monitor this disease:

Creutzfeldt-Jakob disease (CJD) is a rare disease of the central nervous system, which results in death after a relatively rapid course of muscle weakness and dementia. There is no definitive diagnostic test and no known cure.

CJD occurs sporadically in the community with an incidence of one case per million population per year and may have a symptom free incubation period of more than 30 years. CJD is also known to have been transmitted as a result of certain medical interventions, such as human pituitary hormone treatment. This iatrogenic form is similar clinically to sporadically occurring CJD. The symptom free incubation period of this form of CJD ranges from 4 to 30 years with an average of 15 years based on international experience...


For more info visit: http://www.health.gov.au/internet/main/publishing.nsf/Content/health-pubhlth-strateg-phi-index.htm


Creutzfeldt–Jakob disease - From Wikipedia, the free encyclopedia

"Transmissible spongiform encephalopathies (TSEs, also known as prion diseases) are a group of progressive conditions that affect the brain and nervous system of many animals, including humans. According to the most widespread hypothesis they are transmitted by prions, though some other data suggest an involvement of a Spiroplasma infection.[1] Mental and physical abilities deteriorate and myriad tiny holes appear in the cortex causing it to appear like a sponge (hence 'spongiform') when brain tissue obtained at autopsy is examined under a microscope. The disorders cause impairment of brain function, including memory changes, personality changes and problems with movement that worsen over time. Prion diseases of humans include classic Creutzfeldt-Jakob disease, new variant Creutzfeldt-Jakob disease (a human disorder related to mad cow disease), Gerstmann-Sträussler-Scheinker syndrome, fatal familial insomnia and kuru. These conditions form a spectrum of diseases with overlapping signs and symptoms..."


Posted by LYME GREEN AUSTRALIA at Wednesday, September 16, 2009 
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3 comments:

  1. Wow i love you blog its awesome nice colors you must have did hard work on your blog. Keep up the good work. Thanks
    Reply
  2. I soooo made this connection to, though from working with CJD and its symptoms to living Lyme ones... Glad i'm not alone and thank you for this! I can only go through the behavioural patterns I have witnessed in both, now i can see why!
    Reply
  3. the Canadian Consumer Health have an interesting article on mycobacteria: THE LINKING PATHOGEN IN NEURO-SYSTEMIC DISEASES: CHRONIC FATIGUE, ALZHEIMER'S, PARKINSON'S & MULTIPLE SCLEROSIS
    by: Scott, Donald W., M.Sc.
    Reply


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