Atteinte de la borréliose chronique persistante , babesia et bartonella ; le déni est grand , battons nous pour la faire connaitre la borréliose de lyme et ses coinfections ; 8 ans de bi et tri therapies d'antibiotiques auxquels j'ai ajouté une partie du protocole Buhner et autres
More Evidence that Morgellons Disease is an Infection
More Evidence that Morgellons Diseas is an Infection, not a Delusion
"The prestigious F1000 Research journal has published yet another paper linking the controversial Morgellons disease (MD) to a spirochetal bacteria. Until recently, MD was widely believed in the medical community to be purely psychological in origin; however, this latest in a string of new publications strengthens the evidence that sufferers are anything but delusional."
A new paper is published suggesting the stigmatized name "Morgellons" be changed to a more scientifically descriptive name for the condition "Borrelial Dermatitis."
http://www.prweb.com/releases/2013/5/prweb10694837.htm
The medical profession must be careful when making a diagnosis …The consequences of such misdiagnosis include delay in appropriate treatment, worsening of the actual disease process, and punitive labeling of the patient as "psychiatric"…Edward Kilbane, MD
"The prestigious F1000 Research journal has published yet another paper linking the controversial Morgellons disease (MD) to a spirochetal bacteria. Until recently, MD was widely believed in the medical community to be purely psychological in origin; however, this latest in a string of new publications strengthens the evidence that sufferers are anything but delusional."
A new paper is published suggesting the stigmatized name "Morgellons" be changed to a more scientifically descriptive name for the condition "Borrelial Dermatitis."
http://www.prweb.com/releases/2013/5/prweb10694837.htm
The Charles E Holman Foundation Medical Advisor Suggests New Name for Mystery Disease Based on latest Research.
The medical profession must be careful when making a diagnosis …The consequences of such misdiagnosis include delay in appropriate treatment, worsening of the actual disease process, and punitive labeling of the patient as "psychiatric"…Edward Kilbane, MD
London, UK (PRWEB) May 02, 2013
The prestigious F1000 Research journal has published yet another paper linking the controversial Morgellons disease (MD) to a spirochetal bacteria. Until recently, MD was widely believed in the medical community to be purely psychological in origin; however, this latest in a string of new publications strengthens the evidence that sufferers are anything but delusional. Internist/Dermatologist Peter Mayne of Laurieton, NSW, Australia is the principal author of the latest scientific article on MD, Morgellons: a novel dermatological perspective as the multisystem infective disease borreliosis. The co-authors in this international team of collaborators include: Dermatologist Dr. John English from Nottingham University, Nottingham, UK; Psychiatrist Dr. Edward Kilbane from Stanford University, Palo Alto, CA, USA; Jennie Burke, Director of Australian Biologics, Sydney, NSW, Australia; Veterinary microbiologist Marianne J. Middelveen from Calgary, Alberta, Canada; and Internist Dr. Raphael B. Stricker from San Francisco, CA, USA.
The unusual fibers seen in skin lesions of patients with Morgellons Disease are considered the hallmark of the disease and believed by mainstream doctors to be deliberately implanted by patients. Earlier studies on these multicolored fibers showed that they were actually produced in the skin by the patients’ skin cells and composed of the skin proteins collagen and keratin. These studies also found Borrelia spirochetes in the skin lesion. This bacteria is known for causing Lyme disease, thus suggesting that MD may actually be an unusual manifestation of Lyme disease.
This new paper by Mayne and his colleagues paints MD as a multisystem disorder and elaborates by examining an Australian family where three members simultaneously had symptoms of MD. Spirochetes were detected in the skin lesions of two members and one was identified by DNA sequencing as Borrelia garinii, a causative agent of Lyme disease in Europe and Asia.
The new paper also presents a differential diagnosis from a dermatological perspective and an in-depth analysis of the diagnosis of delusional disorders from a psychiatric perspective. The study arduously points out that an infectious disease can produce psychiatric symptoms. The authors propose to de-stigmatize this illness within the medical community by replacing the popular moniker Morgellons disease with a proper medical name, Borrelial Dermatitis.
The CEHF applauds the publication of this preeminent research by volunteer Medical/[Scientific Board members, Peter Mayne, Edward Kilbane, Marianne Middelveen and Raphael Stricker. The Charles E. Holman Foundation (The CEHF), based in Austin, TX, is a 501(c) (3) nonprofit organization committed to advocacy and philanthropy for Morgellons disease. The foundation was named for Charles E. Holman, a pioneer in the fight against Morgellons disease and is led by Executive Director Cindy Casey Holman, RN.
Only those with Lyme Disease know how to pull out all the stops and help support our Lyme heroes and warriors
$12,000 raised in first day of microscope fundraising
2nd May 2013
On the morning of May 1, we explained about the important Lyme disease research conducted by Dr. Eva Sapi and her need for an expensive, highly specialized microscope. We asked you to help us raise the last $20,000 required to procure this instrument and otherwise support her research. In the first 24 hours, you stepped up to the plate and donated more than half that amount. Great job, folks! And let's keep going. If you haven't already, we hope you'll watch the video about Dr. Sapi's work, donate if you can, and help us spread the word by forwarding the link to your family and friends, as well as sharing this on Facebook and Twitter.
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Lyme Disease: Some Thoughts On Its Coinfections by Buhner
http://www.wisdom-magazine.com/Article.aspx/3130/
Lyme Disease: Some Thoughts On Its Coinfections
It has become clear the past few decades that many new infections, and others, simply not recognized as being infections (e.g. endocarditis from bartonella infection or arthritis from lyme bacteria), are emerging with greater frequency and virulence in the human population. Those that belong to the Lyme disease group are some of the more worrisome. This includes, but is not limited to, infections caused by various species of Borrelia burgforferi sensu lato, Babesia, Bartonella, Ehrlichia, Anaplasma, and Mycoplasma, and a number of lesser players (at this point) such as the relapsing fever borrelias, Rocky Mountain Spotted fever (Rickettsia rickettsii), Tick-borne encephalitis, Colorado Tick Fever (coltivirus species), Coxiella, and variousWolbachia species. Factually, this group of organisms should be considered to be an arthropod-spread emerging disease complex of parasitic organisms, not specifically lyme and its coinfections, nevertheless . . .
The Lyme-group of infections presents unique difficulties to most Western approaches, whether community herbalism, reductive phytorationalism, or technological medicine. The organisms are especially good at sequestering themselves inside host cells to avoid immune responses. As well, they affect a wide variety of body sites, leading some researchers, such as Lynn Margulis, to refer to their increasing incidence as the "resurgence of the great imitator."
The stealth nature of the organisms and the complexity of their infectious cycle necessitates a very different approach than is usually common in the Western tradition. The paradigm of a silver bullet, i.e. antibacterials, specific to the invading organisms has proven inadequate. The organisms are exception-ally good at generating a swarm of genetic varients in short time lines, creating subtypes that are unaffected by antibiotics. They often infect difficult to reach areas of the body, places antibiotics cannot easily reach. As well, they infect a wide variety of bodily systems, mimicking in their symptoms, a wide range of disease conditions. Thus the infected have been diagnosed with such things as arthritis, bi-polar disorder, multiple sclerosis, schizophrenia (various types), Alzheimer’s, Parkinson’s disease, heart disease, irritable bowel syndrome, and so on.
A successful treatment, I have found, necessitates a break with traditional approaches. A decade of focus on the organisms has found that the best outcomes consistently occur if the following are used when approaching treatment, specifically understanding:
1) the flexibility and infectious dynamics of the organisms;
2) the synergy that occurs between the organisms;
3) the cytokine cascade the organisms initiate;
4) the need for acute attentiveness to the uniqueness of the infected person, that is, the protocol has to be designed to match the person’s ecological reality and as that alters over time, the protocol consistently needs to be adjusted with a high degree of sensitive awareness;
5) the protocols, to be effective, must accomplish the following:
a) reduce the cytokine cascade;
b) enhance immune function;
c) protect the areas of the body that are targeted;
d) address specific symptoms;
e) involve depth human contact between the practitioner and the client;
f) and understand that a focus on killing the organisms as a first line intervention, with certain caveats, is often the least successful approach.
Pharmaceutical/technological medical approaches are generally weak in these areas. Antibiotics, numerous peer reviewed studies have found, only help about 60% of those infected. While some of the more effective antibiotics such as doxycycline initially show up to a 95% success rate there is, in those populations, about a 30-40% relapse rate. That is one of the primary reasons the lyme community is so involved, much as the AIDS community has been. They have found that a combination of pharm-aceuticals and other approaches, specifically herbal medicines, to be the most effective in finding wellness. It is possible to heal from lyme and coinfections; it just takes work, time, and a great deal of sensitivity.
Unfortunately, you can’t treat lyme without encountering lyme politics. The CDC guidelines on lyme and the various coinfections are incorrect. Nevertheless even they note a yearly increase in infections. In 2002, the incidence was 20,000 per year; it is now closer to 40,000. Harvard researchers, however, put those figures at least ten times higher. And this is only lyme, the disease pool for coinfections is considerably larger. Bartonella and mycoplasma infections are very high.
Genetic Flexibility
All Lyme-group organisms are undergoing rapid genetic alterations in response to environmental factors such as climate change, habitat damage, and human population increase. They are tremendously genetically flexible and they exist within their hosts as a mosaic of genetic variants. It is more accurate to think of them as a self-organized swarm, not as individual microbial organisms. This provides multiple advantages: it maximizes survival of the swarm when attacked by host immune responses and allows discrete elements of the swarm entry into different physiological niches, e.g. bone marrow or lymph system, again maximizing survival. It is not uncommon that several variants can be found within those niches, the several strains exchanging genetic material in order to stay ahead of the immune response. The outer membrane proteins on the exterior of the cell wall are often altered (as are many of the adhesion molecules they use to attach to host cells) which makes the variants harder to recognize by both the immune system and pharmaceuticals. Simple rearrangements of certain portions of the genome can create as many as 420,000 variants of a species of bacteria in a short period of time.
Niche Specifics
Each member of the Lyme-group targets specific areas of the body for infection; each has its preferred habitat. In general, once at the target location, the organisms stimulate inflammation in order to break down target cells, freeing the nutrients they need in order to feed. Lyme spirochetes are specific for collagen tissues and the symptoms of infection appear wherever they break down those tissues. If in the joints, arthritis. If in the heart, lyme carditis, in the central nervous system and brain, neuroborreliosis, in the eye ocular borrelliosis, the meninges of the brain, meningitis. They also adhere to endothelial and epithelium cells, differentiated neural cells, brain cells, and glial cells. Each of these cellular locations can degrade in response.
Babesia infects red blood cells (and the spleen), similarly to malarial organisms; the symptoms are similar. Bartonella infects endothelial cells and red blood cells. Mycoplasma organisms tend to infect red blood cells, the reproductive tract, respiratory system, and gustatory system. They have a tropism for cilia, among other things. Mycoplasma have such a reduced genome that they need to scavenge large numbers of nutrients from the body; they affect a much broader range of body systems.
Immune Dynamics
It is crucial to address immune status as part of any treatment protocol, irrespective of orientation. There is an inescapable truth revealed by research on the Lyme-group: The weaker or more compromised the immune system, the more likely someone is to become infected and the more likely they are to have a debilitating course of illness. Improving the immune status of those infected allows the immune system, refined over long evolutionary time, to do what it does best, i.e. use very elegant mechanisms to control and clear infection.
Ultimately, when supported, the immune system will identify the outer membrane proteins of the microbes and create antibodies to them. Depending on the type of infection, the number of coinfectious agents, and the health of the immune system (and the particular ecology) at time of infection, this can take, in those whose immune system can do the work on its own, anywhere from four to 12 months. In those whose immune systems are compromised it may take longer; how long is directly proportional to the health of the immune system. Once the immune system creates the proper antigens, the bacteria are then eliminated fairly rapidly from the body.
Neural Damage Including
Sensory Gating Deficits
Because of the common tropism for neural cells among the Lyme-group a wide variety of brain and neural structures can be affected. These impacts generate a wide variety of neurological symptoms and psychological difficulties. If the amygdala is infected, there is sometimes the tendency to homicidal rage (common during bartonella infection). If the hippocampus is affected, the ability to determine meaning can be affected (common during borrelia infection). Cognitive defects are common including brain fog, memory difficulties, linear reasoning deficits, and extreme emotional lability. Sensory gating deficits are also, unfortunately, common.
Sensory gating is the process whereby the brain reduces the amount of incoming sensory data to protect the conscious mind from overload and fragmentation. Gating deficits generally increase the amount of sensory data that is perceived, a common experience in schizophrenia or when taking hallucinogens. Every sensory modality has its own neural gating stream; any or all can be affected. Lyme-group infections commonly cause increases in visual, auditory, and somatosensory inputs – and occasionally olfactory and gustatory. In the auditory system, this can cause PTSD symptoms and extreme sensitivity to noise. In the visual, sensitivity to light. In the somatosensory, sensitivity to touch. In the olfactory, multiple chemical sensitivity. For some, there is a feeling of being overwhelmed with sensory inputs, as if the whole world were rubbing against the sensory network.
Reducing inflammation in the brain will help reduce the problems over time. The use of neural regenerators will help restore damaged neural networks. The use of anxiety reducing herbs will help bridge the time period before restoration. Sophisticated psychotherapeutic interventions will reduce the hysteria that often accompanies severe cognitive deficits; the feeling of being accompanied in the darkness does produce significant increases in the degree and rapidity of healing.
Some Useful Herbs
Lyme borreliosis:
Polygonum cuspidatum root. Uncaria tomentosa.
Primary cytokine cascade inhibitors: Scutellaria baicalensis, Cordyceps, Isatis, Houttuynia, Polygonum cuspidatum.
Primary immune herbs:
Glycyrrhiza, Rhodiola, Eleutherococcus, Cordyceps, Withania.
Red blood cell protectants:
Sida acuta, Cryptolepis
Neural regenerators:
Polygala tenifolia root, Hericium erinaceus
Dosage Guidelines
Dosage varies considerably for people with this group of infections. Some people are so hypersensitive that they can tolerate only tiny amounts of herbal intake, from one to five drops at a time. Others need large quantities, up to one teaspoon three to four times daily. For those that can tolerate it, dosages of cytokine herbs should be high in order to flood the body with substances that will keep the cytokine cascade in check. Dosages should start small and then be adjusted for the particular body ecology of the person, down or up as necessary. Ultimately, dosage is an art form, in herbalism and in treating this group of infections. Again, there is no one size fits all approach for this population of clients, nor is there one approach that will help all of them.
WARNING: Herbs can have very powerful medicinal actions in the body and could produce severe and possibly life-threatening interactions with other medications you may be currently taking. Be sure to consult with your regular healthcare provider before taking any herbal medications.
Final Comments
This group of infections can be successfully treated; we just need to develop a more sophisticated approach to doing so. There is no way that reductive, linear approaches can keep up with the nonlinear, self-organized identities that these organisms are. The reductive beginnings are a good foundation, ultimately however, the final treatment interventions occur by feel, by a sense of the art, a deep empathy for the client, all rooted in a deep relationship with the plants and a depth understanding of the organisms as discrete intelligent identities acting in concert with their natures in unique, and never-identical body ecologies.
Stephen Harrod Buhner lectures throughout the United States on herbal medicine, the sacredness of plants, and the intelligence of nature. His new book Healing Lyme Disease Coinfections in being released in May 2013. Hisother books include Healing Lyme, The Secret Teachings of Plants, and The Transformational Power of Fasting. He lives in Silver City, New Mexico.
Lyme Disease Interview 2006/2012
http://www.youtube.com/watch?v=-frg8Evh54w
Publiée le 1 mai 2012
I filmed myself talking about my life in 2006, when I was bedridden due to Lyme Disease, when I was fourteen. I now went back and edited in my thoughts on it now, as a 20 year old.
Catégorie
Licence
Licence YouTube standard
OHIO IS NO LONGER TESTING TICKS FOR DISEASE
In Ohio, recent budget cuts prevent the state from testing ticks for Lyme disease. "Without this data, we won't know how many ticks are carrying Lyme Disease or Rocky Mountain Spotted Fever. Therefore you need to take the prevention into your own hands."
Read more:http://ow.ly/kzGfr
http://www.whiznews.com/content/news/local/2013/04/29/ohio-is-no-longer-testing-ticks-for-disease
Read more:http://ow.ly/kzGfr
http://www.whiznews.com/content/news/local/2013/04/29/ohio-is-no-longer-testing-ticks-for-disease
OHIO IS NO LONGER TESTING TICKS FOR DISEASE
Recently the Ohio Department of Health alerted local health officials that due to state budget cuts, they will no longer be testing various insects for potential diseases.
This poses a potential prevention problem for a rural and wooded areas, such as Muskingum County, where three types of ticks can be found. Without this data, we won't know how many ticks are carrying Lyme Disease or Rocky Mountain Spotted Fever. Therefore you need to take the prevention into your own hands.
"The state would perform the testing in order to determine if the specific tick was carrying a disease that we would be concerned about it spreading to humans,” said Matt Hemmer, a Public Health Sanitarian at the Health Department. “What I would recommend for people, is submit them to your local health department, and we can identify them to species and make recommendations based on that."
All tick bites, contaminated with disease or not, will initially have the same reaction, but you need to pay attention to possible symptoms of each disease.
"Those usually consist of rash, headache, usually a fever, chills, and also reddening of the eyes can be seen also. "
Last year in Muskingum County there was only one case of each Lyme and Rocky Mountain Spotted Fever but the entire state had 93 cases.
Jak skutecznie ochronić psa i kota przed kleszczami?
http://weterynarz-olkusz.pl/2013/jak-skutecznie-ochronic-psa-i-kota-przed-kleszczami/
JakJak skutecznie ochronić psa i kota przed kleszczami? skutecznie ochronić psa i kota przed kleszczami?
Na polskim rynku pojawiają się wciąż nowe preparaty przeciw najczęściej spotykanym pasożytom zewnętrznym, czyli pchłom i kleszczom. Które z nich wybrać i jak je stosować, aby stworzyć maksymalną ochronę naszych czworonogów przeciw chorobom zakaźnym przenoszonym przez kleszcze?
Aby dokonać właściwego wyboru, musimy pamiętać o kilku sprawach.
1. Nie wszystkie preparaty działające na pchły, chronią naszego pupila przed kleszczami. Jednak te, które działają przeciwkleszczowo zapobiegają przed inwazją pcheł. Czytajmy więc ulotkę.
2. Zazwyczaj czas działania prawidłowo założonego preparatu przeciwkleszczowego typu spot-on (do nakraplania na skórę zwierzęcia) to okres 4 tygodni. Obróżki mogą „działać” nawet 7- 8 miesięcy. Co jest niezwykle istotne – aby rzeczywiście czas ochrony naszego zwierzaka był równy temu, jaki podaje producent na ulotce, powinniśmy zwracać uwagę na sposób ich użycia.
3. Podstawowym błędem, jaki właściciele czworonogów popełniają, zwłaszcza stosując preparaty typu spot-on, to kąpiel zwierzaka w zbyt krótkim czasie przed czy też po nakropleniu preparatu.Radzimy założenie preparatu typu spot - on na skórę zwierzaka przed/ po 10 dniach od kąpieli. Większość dostępnych preparatów rozprowadza się w gruczołach łojowych i wydzielanym przez nie sebum.
4. Pamiętajmy, aby aplikować dokładnie taką ilość preparatu, jaka jest przewidziana na nasze konkretnie zwierzę. Zważmy zwierzaka przed zakupem leku. Preparaty spot - on są konfekcjonowane w pipetach do jednorazowego zużycia, o objętości odpowiedniej dla danego przedziału masy ciała zwierzęcia. Dlatego też próba „zaoszczędzenia” środka i podawanie preparatu „po kropelce” skutkuje brakiem jego działania.
5. Jeśli jesteśmy właścicielami kota, zwróćmy szczególną uwagę na postać wybieranego preparatu. Jeśli decydujemy się na wybór obróżki, pamiętajmy, aby założyć ją niezbyt luźno. Zdarzają się nieszczęśliwe przypadki zawieszenia kota - wędrownika na drzewie czy płocie właśnie za taką niedopasowana obróżkę.
6. Jeszcze bardziej istotną kwestią dla właścicieli kotów czy królików jest sprawdzenie, czy dany preparat jest dopuszczony do stosowania u tego gatunku. Duża część środków przeciwkleszczowych jest przeciwwskazana do stosowania u kotów i królików. Ich użycie może skutkować poważnym zatruciem, a nawet śmiercią zwierzęcia. Bezpieczniej skonsultować się w tej sprawie z lekarzem weterynarii.
7. Pamiętając o ochronie naszego środowiska, warto przeczytać dokładnie ulotkę preparatu, który kupiliśmy. Wiele z nich (zarówno obróżki jak i pipety) zawierają substancje toksyczne. Dlatego, jeśli planujemy kąpiel naszego czworonoga w jeziorze czy rzece, sprawdźmy w ulotce, czy nie zaszkodzimy żyjącym tam stworzeniom.
8. I wreszcie – pamiętajmy, że kleszcze czy pchły „uodparniają się” na stosowane przeciw nim preparaty podobnie jak bakterie na antybiotyki. Dlatego obserwujmy naszego zwierzaka, zawsze bowiem może się zdarzyć, że środek używany w poprzednim roku, w tym może okazać się już nieskuteczny.
The Lyme Times , special offer
A $30 donation (includes shipping & handling) will get you 30 Lyme Times for your month of May Lyme Awareness Campaign.
Hand them out at work or school! Give them to your doctors! Put some at the local clinic, farm supply store, or library! Share them with friends and family!
We will send you a mix of several issues (our choice) in a medium USPS flat rate box.
To order, go to LymeDisease.org and click on the Donate Now button in the upper righthand corner.
Scroll down and select Other Amount.
If you donate $30, we will send you 30. If you donate $60, we will send you 60. $90/90 and so on. You can also leave us a message.
Everyone can be an activist!
Hand them out at work or school! Give them to your doctors! Put some at the local clinic, farm supply store, or library! Share them with friends and family!
We will send you a mix of several issues (our choice) in a medium USPS flat rate box.
To order, go to LymeDisease.org and click on the Donate Now button in the upper righthand corner.
Scroll down and select Other Amount.
If you donate $30, we will send you 30. If you donate $60, we will send you 60. $90/90 and so on. You can also leave us a message.
Everyone can be an activist!
Tick-Borne Diseases : ticks may carry other diseases along with Lyme disease.
http://tbdalliance.org/getinformed/other-tbds
OTHER TBDS
Tick-Borne Diseases
While Lyme disease is the most common disease that ticks carry, ticks may carry other diseases along with Lyme disease. All tick-borne disorders tend to include flu-like symptoms of fever, malaise, headache, and joint/muscle aches and pain. These are most common tick-borne diseases:
ANAPLASMOSIS - Caused by the the bacterium Anaplasma phagocytophilium, previously known as human granulocytic ehrlichiosis (HGE) and more recently called human granulocytic anaplasmosis (HGA). Symptons include fever, headache, chills, and muscle aches.
BABESIOSIS - Protozoan infection of red blood cells. Symptoms can be mild to life threatening, with a high fever and fatigue. More severe in people who have had their spleen removed. Strains include:
- Babesia-Microti
- Babesia-WA1
- Babesia divergens
- B. bovis
- Other species are possible
BARTONELLA HENSELAE - CAT-SCRATCH DISEASE - Bacterial disease starting with a red mark that can become swollen and discolored. Symptoms include swollen lymph nodes (especially under ears) with conjunctivitis, heart or spleen problems, bone lesions, hepatitis, eye problems and encephalitis (causing seizures and coma). There is indirect clinical evidence that there are a group of "Bartonella-like organisms" that can co-infect a Lyme patient. Identification of these organisms awaits further scientific study.
COLORADO TICK FEVER - Viral disease characterized by a high fever and sometimes a faint rash. After a 2-3 day remission, symptoms recur, accompanied by a drop in white blood cells. Complications may include encephalitis, heart problems and severe bleeding.
EHRLICHIOSIS - Rickettsial infections (HME and HGE forms) of white blood cells. A rash may occur. Severe illness may have neurologic complications. Delayed treatment can result in death.
LYME (MASTERS') DISEASE - Multisystem bacterial infection that can start with an enlarging rash. Brain, joint, heart, eye, spleen, kidney and other organs can be affected. Lyme disease has been reported worldwide, in every continent except Antarctica. It is well known that migrating birds can distribute ticks long distances, which may be one reason for Lyme's presence all over the globe.
POWASSAN ENCEPHALITIS - Viral brain infection causing seizures, aphasia, muscle weakness, dementia and death.
QUERY (Q) FEVER - Rickettsial infection causing high fever, pneumonia and damage to the liver, heart, or brain. Potential bioterrorism agent because it can become airborne.
RELAPSING FEVER - Multisystem bacterial infection with symptoms similar to Lyme Disease. Characterized by repeating bouts of fever lasting 2-9 days, alternating with periods of no fever. Potential bioterrorism agent.
ROCKY MOUNTAIN SPOTTED FEVER - Rickettsial infection causing a reddish-to-black rash resembling measles. Diagnosis and treatment must be made immediately or death can occur.
TICK PARALYSIS - Loss of motor function and increasing paralysis caused by a reaction to a female tick's neurotoxins.
TULAREMIA - Bacterial infection causing repeated fever spikes. Swollen lymph nodes develop into skin ulcers, conjunctivitis, and pneumonia. Potential bioterrorism agent.
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