As
a clarification: Tick-Borne-Relapsing-Fevers (TBRF) are all caused by
Borrelia bacteria. But instead of being in Hard-Shelled Ticks like Lyme
disease and Borrelia myamotoi, Each Relapsing Fever is associated with
its own species of Ornithidoras "soft-shelled-tick" For example:
Ornithidoras herms ticks contain Borrelia hermsii and causes TBRF and is
associated with a high incidence of MS and chronic-fatigue syndrome
near Lake Tahoe. (See: Ticks and MS by Bonnie Bennet RN who documented the whole CDC debacle in the late 1970s.
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I was a brief acquaintance with Dr. Willy Burgdorfer, and spoke with him weeks before his death. He was trying to tell me and a few others about bad things that he had done early in his American career at the NIH and RML, but he was emotional and hesitant, and withheld certain truths.
Dr. Burgdorfer was a wonderful man and I loved being in his presence, and especially appreciated his clarity in lectures. He never made his research sound erudite.
But for some reason Willy had a guilty conscience?
My opinion is that prior to 1981 microbiologist's like Willy and Dr. Alan Barbour, had already been working with Lyme-like Borrelia species before B. burgdorferi was ever isolated.
I think this is true because Dr. Alan Barbour and others were known to be working with Tick-Borne-Relapsing Fever pathogens.
When they named the Lyme disease organism after Dr. Willy Burgdorfer in 1982, Dr. Alan Barbour became upset that it wasn't named after him. (Why?) Barbour had little to do with Willy's isolate, (at least as far as the world knew.)
A few months after B. burgdorferi is named after Dr Burgdorfer, Dr. Alan Barbour reveals and patents (maybe out of spite) B-31 strain of Borrelia burgdorferi. This laboratory organism immediately became the standard Bb strain for creating Lyme disease serology blood tests.
How did Dr. Alan Barbour do a decade of work in a few months?
To strip two select plasmids from the genomic profile of B. burgdorferi, and then make the bacterium stable, and unchanging despite 1000s of divisions: this takes longer than just a few months ????
Dr. Barbour had to have been keeping this under wraps and secret. But Why?
I think everyone can speculate as to why the government would suppress knowledge of a here-to unknown lab pathogen that could live in Hard-Shelled local ticks and cause human disease.
The question I have is the Ixodes daminii tick species that was first described by Dr Andrew Spielman of Harvard (He has Naval Intelligence background) did he know this "new" tick species could interbreed with Ixodes scapulars local ticks and create viable progeny? Because he said I daminii could not breed successfully with Black legged deer Ticks.
Dr. John Oliver of Southwestern proved him wrong.
I was a brief acquaintance with Dr. Willy Burgdorfer, and spoke with him weeks before his death. He was trying to tell me and a few others about bad things that he had done early in his American career at the NIH and RML, but he was emotional and hesitant, and withheld certain truths.
Dr. Burgdorfer was a wonderful man and I loved being in his presence, and especially appreciated his clarity in lectures. He never made his research sound erudite.
But for some reason Willy had a guilty conscience?
My opinion is that prior to 1981 microbiologist's like Willy and Dr. Alan Barbour, had already been working with Lyme-like Borrelia species before B. burgdorferi was ever isolated.
I think this is true because Dr. Alan Barbour and others were known to be working with Tick-Borne-Relapsing Fever pathogens.
When they named the Lyme disease organism after Dr. Willy Burgdorfer in 1982, Dr. Alan Barbour became upset that it wasn't named after him. (Why?) Barbour had little to do with Willy's isolate, (at least as far as the world knew.)
A few months after B. burgdorferi is named after Dr Burgdorfer, Dr. Alan Barbour reveals and patents (maybe out of spite) B-31 strain of Borrelia burgdorferi. This laboratory organism immediately became the standard Bb strain for creating Lyme disease serology blood tests.
How did Dr. Alan Barbour do a decade of work in a few months?
To strip two select plasmids from the genomic profile of B. burgdorferi, and then make the bacterium stable, and unchanging despite 1000s of divisions: this takes longer than just a few months ????
Dr. Barbour had to have been keeping this under wraps and secret. But Why?
I think everyone can speculate as to why the government would suppress knowledge of a here-to unknown lab pathogen that could live in Hard-Shelled local ticks and cause human disease.
The question I have is the Ixodes daminii tick species that was first described by Dr Andrew Spielman of Harvard (He has Naval Intelligence background) did he know this "new" tick species could interbreed with Ixodes scapulars local ticks and create viable progeny? Because he said I daminii could not breed successfully with Black legged deer Ticks.
Dr. John Oliver of Southwestern proved him wrong.
